Bowel Obstruction

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10 أقسام

Summary

Bowel obstruction is the partial or complete blockage of intestinal lumen impeding passage of contents. Primary causes include adhesions (60-80%) for small bowel obstruction (SBO) and colorectal cancer (50-60%) for large bowel obstruction (LBO).

Clinical features vary by location: SBO presents with colicky periumbilical pain and early bilious vomiting, while LBO shows early pronounced distention with later vomiting. Signs include abdominal distention, tenderness, and altered bowel sounds.

Diagnosis relies on imaging (CT abdomen/pelvis is gold standard) showing dilated proximal bowel, collapsed distal bowel, and multiple air-fluid levels. Laboratory tests assess severity and complications.

Management includes supportive care (IV fluids, nasogastric decompression) with nonoperative approach for simple obstructions. Surgical intervention is indicated for complicated cases (showing strangulation, ischemia), closed-loop obstructions, or when conservative management fails after 72 hours. Complications include bowel ischemia, perforation, and peritonitis.

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Definition

Bowel obstruction refers to the partial or complete blockage of the intestinal lumen that impairs the normal passage of intestinal contents. It is categorized based on:

  • Location: Small bowel obstruction (SBO) vs. large bowel obstruction (LBO)
  • Physiologic mechanism: Mechanical vs. functional (paralytic ileus)
  • Degree: Partial vs. complete
  • Etiology: Intrinsic, intramural, extrinsic, or intraluminal causes
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Epidemiology

  • SBO primary cause: Adhesions (60-80% of cases)
  • LBO primary cause: Colorectal cancer (50-60% of cases) in adults
  • Age distribution: Varies by etiology (see specific causes)
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Etiology

Classification by location and mechanism

Classification by location and mechanism
Classification Description
Dynamic (mechanical)
  • The onset can be acute or chronic
  • The site can be high (small bowel obstruction or low (large bowel obstruction)
  • It can be simple or strangulated (complicated)
Adynamic (paralytic/neurogenic ileum)
  • There is no peristalsis
  • Can be associated with surgery

Classification by location of obstruction

Classification by location of obstruction
Location Common Causes
Inside the lumen Fecal impaction, gallstone ileus, foreign bodies, intussusceptions, parasites (Ascaris lumbricoides)
In the wall Tumors, strictures (Crohn's disease), congenital atresia, diverticulitis
Outside the wall Adhesions, strangulated hernias, volvulus, bands, masses

 

Most common causes by age group

Common Causes of Intestinal Obstruction by Age Group
Age Group Common Causes
Neonatal Congenital anomalies, meconium ileus, Hirschsprung's disease
Infants Intussusception, Hirschsprung's disease, obstruction due to meckel’s diverticulum, strangulated hernia
Young adults Strangulated hernia, Crohn's disease, bands, adhesions
Adults & Elderly Cancer, sigmoid volvulus, diverticulitis, strangulated hernias

 

Small bowel obstruction (SBO)

  • Post-surgical adhesions: 60-80% of cases
    • Risk increases with number of previous abdominal surgeries
    • May occur weeks to decades after surgery
  • Hernias: 10-15% of cases (inguinal, femoral, umbilical, incisional)
  • Neoplasms: 5-10% of cases (primary or metastatic)
  • Crohn's disease: Strictures or inflammatory masses
  • Intussusception: More common in children (90%) than adults
  • Gallstone ileus: More common in elderly women

 

Large bowel obstruction (LBO)

  • Colorectal cancer: 50-60% of cases
    • Most commonly in sigmoid colon and rectum
  • Diverticular disease: 10-20% of cases
  • Volvulus: 10-15% of cases
    • Sigmoid volvulus: More common in elderly, chronic constipation
    • Cecal volvulus: More common in younger patients
  • Strictures: Post-inflammatory, radiation-induced, ischemic
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Clinical presentation

General clinical presentation of intestinal obstruction

  • Abdominal pain
  • Distension
  • Vomiting
  • Absolute constipation

 

Clinical presentation according to the site

High small bowel obstruction

  • Periumbilical pain
  • Early profuse vomiting with rapid dehydration
  • Minimal distension
  • No air fluid levels on abdominal X-ray

 

Low small bowel obstruction

  • Periumbilical pain
  • Delayed vomiting
  • Central distension multiple central air fluid level

 

Large bowel obstruction

  • Distension is early and pronounced
  • Pain is mild
  • Vomiting and hydration are late
  • Distended proximal colon and cecum on abdominal X-ray

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Physical examination

  • Signs of dehydration
  • Visible peristalsis might be present
  • Scars might suggest adhesions and hernias might suggest strangulation
  • Abdominal distension and tenderness might be present

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Small bowel obstruction (SBO)

Background

  • Small bowel obstruction (SBO) indicates partial or complete blockage of the small intestines
  • Risk factors include prior abdominal surgery (most common cause, masses/malignancy (common in elderly), hernias, inflammatory bowel disease, and intussusception (in children)

 

Clinical presentation

  • Symptoms
    • Nausea and vomiting
    • Abdominal pain
  • Physical signs
    • Abdominal distention
    • Generalized tenderness

 

Diagnosis

  • Labs
    • Lactic acid (to monitor for bowel necrosis)
  • Imaging
    • Abdominal X-ray (best initial test) that would show dilated loops of small bowel
    • CT scan of the abdomen and pelvis would show dilated loops of small bowel and any masses present

 

Differential diagnosis

  • Acute appendicitis (imaging would reveal an enlarged appendix with signs of inflammation instead of dilated loops of bowel)

 

Treatment

  • Conservative management
    • IV fluids
  • Medical management
    • Treat the underlying condition
    • Nasogastric decompression and bowel rest (indicated for partial SBO with no signs of bowel strangulation)
  • Surgical management
    • Should be performed to relieve the obstruction and correct the cause of SBO (eg, removal of adhesions and bands)
    • Surgical management is indicated for peritonitis and signs of bowel strangulation (systemic signs, metabolic acidosis and continuous pain)

 

Complications

  • bowel necrosis
  • peritonitis
  • bowel perforation

 

Prognosis

  • Partial SBO is often self resolving
  • Complete SCO may be self resolving but often may require surgical intervention

 

Mind maps

 

Small Bowel Obstruction - Summary
Clinical Presentation
  • Colicky abdominal pain, vomiting
  • Inability to pass flatus or stool if complete (no obstipation if partial)
  • Hyperactive then subsequently absent bowel sounds
  • Distended and tympanitic abdomen
Diagnosis
  • Dilated loops of bowel with air-fluid levels on plain film or CT scan
  • Partial: air in colon
  • Complete: transition point (abrupt cutoff), with no air in colon
Complications
  • Ischemia/necrosis (strangulation)
  • Bowel perforation
Management
  • Bowel rest, nasogastric tube suction, intravenous fluids
  • Surgical exploration for signs of complications
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Large bowel obstruction (LBO)

Background

  • Large bowel obstruction (LBO) is a surgical emergency and requires intervention (obstruction may be partial or complete)
  • LBO is commonly seen in elderly patients
  • Chronic constipation is a major risk factor

 

Clinical presentation

  • Symptoms
    • Cramps abdominal pain
    • Nausea and vomiting
    • Bloating
  • Physical signs
    • Abdominal distention
    • Generalized tenderness

 

Diagnosis

  • Labs
    • Lactic acid (to monitor for bowel necrosis)
  • Imaging
    • Abdominal X-ray (best initial test) that would show dilated bowel (screen for free air under the diaphragm)
    • CT scan of the abdomen and pelvis with contrast (imaging of choice for diagnosis of LBO) to distinguish between a partial or complete obstruction
    • Contrast radiography with enema (bird’s beak appearance in volvulus, Apple core sign in colonic malignancy)
        

 

Differential diagnosis

  • Small bowel obstruction (dilated loops of small bowel seen on imaging rather than dilated loops of large bowel)
  • Ogilvie syndrome (no mechanical lesion on CT imaging)

 

Treatment

  • Conservative management
    • Modified diet (high fiber diet, stool softeners)
  • Medical management
    • IV fluids
    • Nasogastric decompression and bowel rest (indicated for cases with abdominal distention or vomiting)
  • Surgical management
  • Exploratory laparotomy (indicated for complete LBO, bowel ischemia, or volvulus)

 

Complications

  • Ischemic colitis
  • Bowel perforation

 

Prognosis

  • If treated early, mortality for LBO is low
  • If there is bowel ischemia or perforation, mortality is higher Mind maps

 

Mind maps

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Acute Colonic Pseudo-Obstruction (Ogilvie Syndrome)

Acute Colonic Pseudo-Obstruction (Ogilvie Syndrome)
Etiologies
  • Major surgery, traumatic injury, severe infection
  • Electrolyte derangements (hypokalemia, hypomagnesemia, hypocalcemia)
  • Medications (e.g., opiates, anticholinergics)
  • Neurological disorders (e.g., dementia, stroke)
Clinical Findings
  • Abdominal distention, pain, obstipation, vomiting
  • Tympanic to percussion, reduced bowel sounds
  • If perforation occurred: guarding, rigidity, rebound tenderness
Imaging
  • X-ray: colonic dilation, normal haustra, nondilated small bowel
  • CT scan: colonic dilation without anatomic obstruction
Management
  • NPO, nasogastric/rectal tube decompression
  • Neostigmine if no improvement within 48 hours
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Sigmoid volvulus

Sigmoid Volvulus
Risk Factors
  • Sigmoid redundancy (e.g., dilation/elongation from chronic constipation)
  • Colonic dysmotility (e.g., underlying neurological disorder)
Clinical Presentation
  • Slowly progressive abdominal discomfort/distention +/- obstructive symptoms (e.g., nausea, emesis, obstipation)
  • Abdomen distended and tympanitic to percussion
Imaging
  • X-ray: dilated, inverted, U-shaped loop of colon (coffee bean sign)
  • CT scan: dilated sigmoid colon, mesenteric twisting (whirl sign)
Management
  • Endoscopic detorsion (e.g., flexible sigmoidoscopy) and elective sigmoid colectomy
  • Emergency sigmoid colectomy if perforation/peritonitis present
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